Editorial Commentary
Proteomic analysis of PAX8 alterations provides new insights into its role as a master regulator of migration in high-grade serous ovarian cancer
Abstract
High-grade serous ovarian cancer (HGSOC) is the most common subtype of ovarian cancer (1). This cancer is a heterogeneous disease associated with few shared genetic mutations (BRCA1, BRCA2, p53) (2) and with widely accepted origination from the fallopian tube epithelium (FTE).
