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Dysfunctional γδ T cells: a contributing factor for clinical tolerance to malaria?

   
@article{ATM6417,
	author = {Emily M. Eriksson and Louis Schofield},
	title = {Dysfunctional γδ T cells: a contributing factor for clinical tolerance to malaria?},
	journal = {Annals of Translational Medicine},
	volume = {3},
	number = {Suppl 1},
	year = {2015},
	keywords = {},
	abstract = {γδ T cells are a small subset of T cells that can rapidly recognize and respond to antigen in a non-MHC restricted manner. The importance of γδ T cells during malaria infection is established by studies in both mouse models and with human cells, where γδ T cells expand during acute blood stage infections (1,2) and control parasitemia (3). We and others have demonstrated that human γδ T cells are one of the predominant cytokine producers following stimulation with Plasmodium-infected red blood cells in vitro (4-6) and following infection (7,8). γδ T cell cytokine responses are also associated with severe disease (9).},
	issn = {2305-5847},	url = {https://atm.amegroups.org/article/view/6417}
}