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Von Hippel-Lindau: implications in development and disease—response

   
@article{ATM31817,
	author = {Kasey R. Cargill and Sunder Sims-Lucas},
	title = {Von Hippel-Lindau: implications in development and disease—response},
	journal = {Annals of Translational Medicine},
	volume = {8},
	number = {4},
	year = {2019},
	keywords = {},
	abstract = {The authors would like to thank Park et al. for their insightful editorial commentary on our recently published article titled “Von Hippel-Lindau Acts as a Metabolic Switch Controlling Nephron Progenitor Differentiation” (1). The commentary provides an excellent overview of the VHL/HIF axis and its role in cellular oxygen sensing. The editorial authors correctly imply that our study revealed a novel role for VHL in kidney development such that nephron progenitors are required to undergo a metabolic switching event. This reprogramming from glycolysis to mitochondrial respiration was also found to be critical for adequate nephron formation (1).},
	issn = {2305-5847},	url = {https://atm.amegroups.org/article/view/31817}
}