Article Abstract

Mind the gap: renal tubule responses to injury and the role of Cxcl12 and Myc

Authors: Veronika Sander, Richard W. Naylor, Alan J. Davidson

Abstract

Acute kidney injury (AKI) is a common disorder resulting from multi-factorial ischemic, toxic and septic insults to the kidney (1,2). A common feature of all of these injuries is that there is often a profound reduction in renal blood flow resulting in tissue hypoxia.

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